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Marijuana Inhibits Neuro-degeneration In Animal Model Of Multiple Sclerosis,
Study Says
London, United Kingdom: Cannabinoids and the cannabinoid receptor system offer neuroprotection against allergic encephalo myelitis (EAE), an animal model of Multiple Sclerosis (MS), according to findings published in the July 22, 2003 issue of the journal Brain.
Return Back To Main Medical Reports Page Scientists at London's Institute of Neurology determined that mice deficient in the cannabinoid receptor CB1 developed "substantial neurodegeneration" as a result of EAE Researchers also noted that "exogenous CB1 agonists (agents that bind to the receptor, such as THC)can provide significant neuroprotection from the consequences of inflammatory CNS disease in an experimental ... model." Authors concluded: "Therefore, in addition to symptom management, cannabis may also slow down the neurodegenerative processes that ultimately lead to chronic disability in multiple sclerosis and probably other diseases." Multiple sclerosis is believed to be a neurodegenerative disease that is triggered by an inflammatory attack of the central nervous system. Although several previous human studies have demonstrated that marijuana may provide symptomatic relief to common symptoms of MS such as muscle spasms, depression and incontinence, the U.K. study is one of the first to indicate that cannabinoids may potentially stave the onset of the disease. A previous study published in the May 6, 2003 issue of the journal NeuroReport similarly noted that "cannabinoids could provide neuroprotection" and "modify neurodegeneration in Huntington's disease." For more information, please contact Paul Armentano, NORML Senior PolicyAnalyst, at (202) 483-5500. Abstracts of both studies, "Cannabinoids inhibit neurodegeneration in models of multiple sclerosis" and "Effects of cannabinoids in the rat model of Huntington's disease generated by an intrastriatal injection of malonate," are available online via the PubMed search engine at: http://www.ncbi.nlm.nih.gov/PubMed/ <http://www.ncbi.nlm.nih.gov/PubMed/> ------------------------------------------------------------------------ Brain. 2003 Jul 22 [Epub ahead of print]. Cannabinoids inhibit neurodegeneration in models of multiple sclerosis. Pryce G, Ahmed Z, Hankey DJ, Jackson SJ, Croxford JL, Pocock JM, Ledent C, Petzold A, Thompson AJ, Giovannoni G, Cuzner ML, Baker D. Department of Neuroinflammation, Institute of Neurology, University College London, London, UK. Multiple sclerosis is increasingly being recognized as a neurodegenerative disease that is triggered by inflammatory attack of the CNS. As yet there is no satisfactory treatment. Using experimental allergic encephalo myelitis (EAE), an animal model of multiple sclerosis, we demonstrate that the cannabinoid system is neuroprotective during EAE. Mice deficient in the cannabinoid receptor CB1 tolerate inflammatory and excito toxic insults poorly and develop substantial neurodegeneration following immune attack in EAE. In addition, exogenous CB1 agonists can provide significant neuroprotection from the consequences of inflammatory CNS disease in an experimental allergic uveitis model. Therefore, in addition to symptom management, cannabis may also slow the neurodegenerative processes that ultimately lead to chronic disability in multiple sclerosis and probably other diseases. PMID: 12876144 [PubMed - as supplied by publisher] Return Back To Main Medical Page
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