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Cannabinoid May Halt Alzheimer's Progression, Study
Says
La Jolla, CA: THC inhibits the formation of amyloid plaque, the primary marker
for Alzheimer's disease (AD), far more effectively than approved medications,
according to preclinical data to be published in the journal Molecular
Pharmaceutics.
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August 2006
Investigators at The Scripps Research Institute in La Jolla, California reported
that THC inhibits the enzyme responsible for the aggregation of amyloid plaque in a
manner "considerably superior" to approved Alzheimer's drugs such as donepezil and
tacrine.
"Our results provide a mechanism whereby the THC molecule can directly impact
Alzheimer's disease pathology," researchers concluded. "THC and its analogues may
provide an improved therapeutic [option] for Alzheimer's disease [by]...
simultaneously treating both the symptoms and the progression of [the] disease."
Previous studies have shown cannabinoids to possess anti oxidant and
anti-inflammatory properties, both of which may play a role in moderating
Alzheimer's.
Last year, investigators at Madrid's Complutense University and the Cajal Institute
in Spain reported that the intracerebroventricular administration of the synthetic
cannabinoid WIN 55,212-2 prevented cognitive impairment and decreased neurotoxicity
in rats. Other cannabinoids were also found to reduce the inflammation associated
with Alzheimer's disease in human brain tissue in culture. "Our results indicate
that... cannabinoids succeed in preventing the neurodegenerative process occurring
in the disease," investigators concluded.
Over 4.5 million Americans are estimated to be afflicted with Alzheimer's disease.
That figure is expected to triple over the next 50 years.
Previous human trials of synthetic THC (Marinol) and Alzheimer's found that
administration of the drug reduced agitation and stimulated weight gain in patients
suffering from the disease.
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